缅北强奸

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• Researcher,听Douglas Research Centre,听Neuromolecular and brain cell specific underpinnings of mood disorders
• Member,听缅北强奸 Group for Suicide Studies
• Assistant Professor,听Department of Psychiatry, 缅北强奸
• Member of the听Quebec Network for Suicide, Mood Disorders, and Related Disorders ()
• RQSHA co-director of the听Douglas Bell Canada Brain Bank ()
• Principal Investigator at the Ludmer Centre for Neuroinformatics and Mental Health

Corina Nagy, PhD, joined the Douglas Institute and the 缅北强奸 Group for Suicide Studies in 2020, where she has been developing several innovative lines of research exploring听the intricate molecular biology underlying psychiatric phenotypes, with a primary focus on major depression. At the forefront of cutting-edge technology, the aim is to embark on an unprecedented exploration of the brain, achieving an unparalleled level of resolution. The Nagy lab seeks to dissect the contribution of individual brain cells to the pathological state while also tracking the dynamic development of cell-specific gene programs over time in a sex-dependent manner.听

Additionally, is dedicated to studying the communication between the peripheral and central nervous systems. This is achieved by profiling extracellular听vesicles听(EVs) traveling between the periphery and the brain. These multifarious molecules can act as a window into the brain, they can become a therapeutic packaging system, or simply act as a messenger.听听

Through these diverse approaches, the Nagy lab aims to gain a comprehensive understanding of the intricate interactions between different systems, shedding light on the complexity of psychiatric conditions. Prof. Nagy is also a member of Quebec Network for Suicide, Mood Disorders, and Related Disorders (), where she is serves as the Co-director of the Douglas Bell Canada Brain Bank ().

Prof. Corina's area of expertise is Single-cell omics of the human brain and EV profiling from brain and blood. Her laboratory explores different themes, such as:听

• Cell-specific underpinnings of sexual dimorphism in depression;
• Extracellular vesicle mediation of brain communication and plasticity;
• Cell-specific response to pharmacological treatment in depression.

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• Visit for a list of publications.听

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